Endothelial activation by aPL: a potential pathogenetic mechanism for the clinical manifestations of the syndrome

J Autoimmun. 2000 Sep;15(2):237-40. doi: 10.1006/jaut.2000.0412.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Animals
  • Antibodies, Antiphospholipid / metabolism
  • Antibodies, Antiphospholipid / physiology*
  • Antiphospholipid Syndrome / immunology*
  • Antiphospholipid Syndrome / pathology*
  • Antiphospholipid Syndrome / physiopathology
  • Endothelium, Vascular / immunology*
  • Endothelium, Vascular / pathology*
  • Endothelium, Vascular / physiopathology
  • Glycoproteins / immunology
  • Humans
  • beta 2-Glycoprotein I

Substances

  • Antibodies, Antiphospholipid
  • Glycoproteins
  • beta 2-Glycoprotein I